Steven Seidner, M.D.
Professor of pediatrics
Deputy Chairman, University Health System Affairs
Chief of the Division of Neonatology
Medical Director, NICU and Newborn Services
Phone: 210-567-5229
Fax: 210-567-5169
E-mail: seidner@uthscsa.edu
Education:
M.D., University of Arizona Medical School, 1982
B.A., chemistry, Cornell University, 1976
Training:
Fellowship, neonatology, Harbor-UCLA Medical Center, Los Angeles, 1985-88
Residency, pediatrics, Harbor-UCLA Medical Center, Los Angeles, 1982-85
Board Certification:
Pediatrics, 1987; Neonatal-Perinatal Medicine, 1987
Recent Publications:
Vasquez MM, Castro R, Seidner SR, Henson, BM, Ashton, DJ, Mustafa SB. Induction of Serum and Glucocorticoid Induced Protein Kinase-1 (sgk1) and Epithelial Sodium Channels (ENaC) by cAMP in Epithelial Cells 2006 Jan. p. S280. (J Invest Med; vol. 54, no. 1).
Biela, TA, Henson, BM, Seidner SR, Castro R, Mustafa SB. Synthetic Glucocorticoids (GC) Selective for Transactivation stumulate alpha-ENaC Formation but do not Suppress Expression of Glucocorticoid Receptors (GCR) in Mouse Lung Epithelial (MLE-12) Cells 2005. (Pediatr Res).
Isaac J, DiGeronimo RJ, Henson BM, Dixon P, Seidner SR, Mustafa SB. Mechanical Stretch stimulates alpha-Epithelial Na+ Channel Expression in Murine Lung Epithelial Cells: Role of MAPKS 2005. p. S292. (J Invest Med; vol. 53).
Interests:
Surfactant Replacement Therapy; Bronchopulmonary Dysplasia; Antenatal and Postnatal Glucocorticoids; Patent Ductus Arteriosus; Congenital Chylothorax; Hypoxemic Respiratory Failure and Inhaled Nitric Oxide Therapy, Molecular physiology of mechanical ventilation; glucocorticoid and cAMP regulation of ENaC and surfactant proteins; cytokine regulation of ENaC signal transduction in the developing lung; regulation of ductus arteriosus remodeling by nitric oxide and prostaglandins; augmentation of surfactant therapy with antenatal and postnatal lung maturational agents; glucocorticoid effects on lung function; lung development; surfactant metabolism, distribution, and function.
